Sunday, November 8, 2009

Atherosclerosis and Iodine Deficiency

The history of atherosclerosis and iodine deficiency is an interesting story. It starts with, such as the cholesterol hypothesis came.

The idea that atherosclerosis is caused by too much cholesterol in the diet began with a study of rabbits. In 1911, two Russian scientists rabbits fed a high cholesterol diet, and reported that after examining the arteries of the rabbits, they found severe arterial plaque developed.

More interestingly, in 1933, researchersrepeated these experiments rabbits cholesterol but decided to add different substances to see if they could prevent atherosclerosis. They divided the rabbits into several groups:

All the rabbits were fed a high cholesterol diet, which in the high cholesterol levels resulted in blood. The control group of rabbits was only given the high cholesterol diet. Experimental group 1: diet plus dried whole thyroid gland.Experimental Group 2: The diet plus thyroxine or T4(valium). Experimental group 3: The diet plus potassium iodide.Experimental Group 4 a.m. to 5 p.m.: diet plus potassium bromide and potassium chloride.

The results from each group were intriguing:

Cholesterol-fed rabbits developed aortic control marked atherosclerosis.Rabbits fed the cholesterol-rich diet and thyroxine (T4) showed mild to moderate aortic atherosclerosis. Rabbits fed cholesterol-rich diet, and either dried up or had no thyroid iodineatherosclerotic lesions. In the group of rabbits of potassium bromide or potassium chloride and cholesterol, all of the rabbits exhibited marked atherosclerosis. In other words, the efficacy of potassium iodide was not shared by potassium chloride or potassium chloride, then potassium was not a mitigating circumstance.

The study showed that the presence of the whole thyroid or iodine led to a significant decline in the development ofAtherosclerosis.

Currently, coronary heart disease the leading cause of death in the United States. The findings from the study may point to a lack of iodine in the American diet as a factor in this mortality.

Today, in the United States and around the world, iodine deficiency is a growing problem. In 1970, only 2.6% of the U.S. population is iodine deficiency. By 2005 this figure had risen to 16.5%.

This is inPart of the federal government and the medical staff to advise the American public to reduce consumption of salt. Since iodized salt is one of the main sources of iodine in the Western diet is, the effect of iodine as well as to reduce consumption.

Other sources of iodine deficiency is the reduction or elimination of iodine in the commercial bakery and dairy products are available in the late 60s and 70s in context.

In addition, bromine, which is toxic,People who will disrupt and replace it with iodine in the thyroid. The levels of bromide ingestion and exposure in the U.S. have increased sharply in the last 30 years.

For example, bromine in antibacterial agents, pesticides and fumigants can be found, and these substances are found in 40% of our food supply. The EPA says that the amount of bromine has increased in human breast milk 10 times in the last ten years.



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