Atherosclerosis and Iodine Deficiency

The history of atherosclerosis and iodine deficiency is an interesting story. It starts with, such as the cholesterol hypothesis came.

The idea that atherosclerosis is caused by too much cholesterol in the diet began with a study of rabbits. In 1911, two Russian scientists rabbits fed a high cholesterol diet, and reported that after examining the arteries of the rabbits, they found severe arterial plaque developed.

More interestingly, in 1933, researchersrepeated these experiments rabbits cholesterol but decided to add different substances to see if they could prevent atherosclerosis. They divided the rabbits into several groups:

All the rabbits were fed a high cholesterol diet, which in the high cholesterol levels resulted in blood. The control group of rabbits was only given the high cholesterol diet. Experimental group 1: diet plus dried whole thyroid gland.Experimental Group 2: The diet plus thyroxine or T4(valium). Experimental group 3: The diet plus potassium iodide.Experimental Group 4 a.m. to 5 p.m.: diet plus potassium bromide and potassium chloride.

The results from each group were intriguing:

Cholesterol-fed rabbits developed aortic control marked atherosclerosis.Rabbits fed the cholesterol-rich diet and thyroxine (T4) showed mild to moderate aortic atherosclerosis. Rabbits fed cholesterol-rich diet, and either dried up or had no thyroid iodineatherosclerotic lesions. In the group of rabbits of potassium bromide or potassium chloride and cholesterol, all of the rabbits exhibited marked atherosclerosis. In other words, the efficacy of potassium iodide was not shared by potassium chloride or potassium chloride, then potassium was not a mitigating circumstance.

The study showed that the presence of the whole thyroid or iodine led to a significant decline in the development ofAtherosclerosis.

Currently, coronary heart disease the leading cause of death in the United States. The findings from the study may point to a lack of iodine in the American diet as a factor in this mortality.

Today, in the United States and around the world, iodine deficiency is a growing problem. In 1970, only 2.6% of the U.S. population is iodine deficiency. By 2005 this figure had risen to 16.5%.

This is inPart of the federal government and the medical staff to advise the American public to reduce consumption of salt. Since iodized salt is one of the main sources of iodine in the Western diet is, the effect of iodine as well as to reduce consumption.

Other sources of iodine deficiency is the reduction or elimination of iodine in the commercial bakery and dairy products are available in the late 60s and 70s in context.

In addition, bromine, which is toxic,People who will disrupt and replace it with iodine in the thyroid. The levels of bromide ingestion and exposure in the U.S. have increased sharply in the last 30 years.

For example, bromine in antibacterial agents, pesticides and fumigants can be found, and these substances are found in 40% of our food supply. The EPA says that the amount of bromine has increased in human breast milk 10 times in the last ten years.

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Iodine and the Importance in Nutrition

The chief treasure of iodine in the body is the thyroid gland. The main thyroxine, which is secreted by this gland, is made by the circulating iodine. Thyroxine is a wonder chemical which controls the basic metabolism and oxygen consumption of tissues. It increases the heart rate as
and urinary calcium excretion. Iodine regulates the rate of energy production and body weight and promotes proper growth. It improves the spiritual zeal and promotes a healthy Hair, nails, skin and teeth.

The best dietary sources of iodine are kelp and other algae. Other good sources are turnip
Vegetables, garlic, watercress, pineapples, pears, artichokes, citrus fruits, egg yolks and shellfish
and cod liver oil. The recommended dietary allowances are 130 mcg. per day for adult men
and 100 mcg. per day for adult women. An increase to 125 mcg. per day during pregnancy and
mcg to 150. not recommended per day during lactation was.> Deficiency can cause goiter and
Enlargement of the thyroid.

Small doses of iodine are of great value in the prevention of goiter in areas where it is endemic,
and the value in treatments, at least in the initial phase. Larger doses have a temporary value
In the preparation of patients with hyperthyroidism for surgical intervention.

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Causes of Thyroid Goiter and Iodine Treatment

A goiter is an enlargement of the thyroid. The thyroid gland is at the base of the neck. It secretes a fluid that the hormone thyroxine, which regulates from iodine and tyrosine, an amino acid from s. The thyroid gland by the hormone thyroxine metabolism, or burning of food, growth and body temperature and affect mental and emotional balance, it is also a factor in the functioning of the reproductive system.

Thyroid disorders are caused by an inadequate intakeIodine, which is not present in sufficient thyroxine production or a failure to elsewhere in the body that needs to produce more thyroxine than the gland. Goiter can be caused by a lack of iodine in the diet, inflammation of the thyroid gland due to infection or under - or over-production of thyroxine of the thyroid gland.

The symptoms of goiter is a swelling at the base of the throat, hoarseness, change in the rate of metabolism, and in extreme cases, difficulties inSwallowing and breathing.

The treatment of goiter vary with the cause. If goitre is due to a lack of iodine, so that the intake of iodine is a further enlargement of the gland to prevent, reduce and in some cases, their size.

Vitamin A is essential for the proper metabolism of iodine, this vitamin is also essential for the proper functioning of the pituitary gland, a substance that regulates the thyroid gland secretes.

Tyrosine can notwithout vitamin B6 and C. Vitamin E intake of iodine. The increased use of iodized salt has helped to eliminate goiter in many places, where iodine does not occur naturally in foods.

Kelp is an excellent source of iodine, low sodium and is better retained by the body as potassium iodide.

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Iodine Deficiency Can Cause Cretinism - Prevent Cretinism From Happening to Your Child

Cretinism, for those who may not be really aware, shows insidiously in a child. Some parents might not notice the signs easily in their children. However, if detected and treated in time, you can curb Cretinism stop and give your child a better chance at a happier life.

What is cretinism?

Cretinism is a condition that is caused by the extreme hypothyroidism during fetal life, infancy and childhood. The characteristics of cretinism include body atrophyGrowth and mental retardation. Hypothyroidism during the critical stages of development is very devastating to the central nervous system. Severe hypothyroidism in a fetus or a child could lead to mental retardation, one of the most devastating effects of cretinism. However, recognize early signs of hypothyroidism could prevent the permanent features of cretinism.

What are the causes cretinism?

Cretinism is the result of a congenital deficiency of thyroid diseaseGland. This condition is also known as congenital cretinism. It may come from the inability of the thyroid gland, sufficient amounts of thyroid hormones, which are the development of a child due to a genetic defect is crucial to produce the thyroid gland itself.

The most preventable cause of cretinism is a lack of iodine in the diet. This type of cretinism is endemic cretinism. In contrast to congenital cretinism varied, the severity of endemic cretinism in the amount ofIodine in the diet. In some places where the soil is iodine deficiency, and sources of iodine are scarce cretinism whole populations have been observed trends.

Iodine, thyroid function, and cretinism

The mechanisms for the effect of thyroid hormones on the development of people are still not largely explains. However, it was found that a severe lack of thyroid hormone in fetal and child development stagesis largely devastating effect on the central nervous system development of the child.

Iodine is a trace mineral that is necessary for normal thyroid function. Small amounts of iodine in the bloodstream are trapped by the thyroid gland, which produce two hormones, which determine most of the metabolic rate of the entire body.

With a shortage of either iodine or thyroid hormone, the child is suffering from hypothyroidism, and in extreme casesCases of cretinism. Mental and skeletal growth is much slower compared to the soft tissue growth. If the child or infant is not diagnosed within a few weeks of mental disability is permanent.

Detect Hypothyroidism in children

Newborns can still be normal by the presence of maternal thyroid hormones in their bodies. But a few weeks after the birth of the baby's movements may become sluggish and start both the physical and mental growth, may be delayed. The babymay also apply for a longer time to sleep and the noise is weaker than most. Some parents may think that this quiet behavior in their children that they have a well-behaved child. It is always advisable to be cautious and consult a doctor immediately.

Hypothyroid infants can be identified either be treated with iodine if they are suffering from the early stages of endemic cretinism or thyroxine if the thyroid is damaged, is absent or unable to produce the necessary quantities of thyroidHormone. Treatment at any time usually leads to a return to a normal physical growth, but after a few weeks, despite treatment of mental retardation is already permanent.

Endemics cretinism can be prevented if the mother of foods eaten with adequate amounts of iodine. In some developed countries endemic cretinism has been eliminated by correcting the iodine deficiency in expectant mothers and their children. However, some cases of hypothyroidism, yet seemnow and again. It is for this reason that many countries now recognize the screen newborns for hypothyroidism, the full syndrome, before it is too late.